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dc.contributor.author | Cuevas González, Juan Carlos | es |
dc.contributor.author | Vega Memije, María Elisa | es |
dc.contributor.author | Garcia-Vazquez, Francisco Javier | es |
dc.contributor.author | Rodríguez Lobato, Erika | es |
dc.contributor.author | Farfan-Morales, José-Eduardo | es |
dc.date.accessioned | 2016-04-06T12:31:13Z | |
dc.date.available | 2016-04-06T12:31:13Z | |
dc.date.issued | 2016 | es |
dc.identifier.citation | Cuevas González, Juan Carlos ; Vega Memije, María Elisa ; Garcia-Vazquez, Francisco Javier ; Rodríguez Lobato, Erika ; Farfan-Morales, José-Eduardo. Apoptosis and apoptotic pathway in actinic prurigo by immunohistochemistry. En: Medicina oral, patología oral y cirugía bucal. Ed inglesa, 2016, Vol. 21, No. 1: 53- | es |
dc.identifier.uri | http://hdl.handle.net/10550/52016 | |
dc.description.abstract | Background: Actinic prurigo (AP) is an idiopathic photodermatosis, this entity requires exposure to UV-B and -A to develop lesions. Apoptosis is a physiological death program that can be initiated by a permanently active mechanism (extrinsic pathway) or irreparable damage (intrinsic pathway). Material and methods: Descriptive study, the sample size comprised 64 paraffin blocks of tissue with a diagnosis of AP. In H&E-stained slides, the diagnosis of AP was corroborated, and 1-µm-thick sections were processed for immunohistochemistry (IHC). A database was constructed with SPSS version 20, Inc., Chicago, IL, USA, and descriptive statistics were analyzed by X2 test and comparison of means. Results: A total of 64 cases were processed, of which 40 (62.5%) were cheilitis AP and 24 (37.5%) were AP in the skin. Of the 40 cheilitis samples, 27 were positive for Bcl-2 and caspase 3 (67.5%), p53 was expressed in 30 (75%). Of the skin lesions, p53 and caspase 3 were expressed in 18 of 24 cases (75%), and 13 were positive for Bcl-2 (54%). Conclusions: We propose that apoptosis is the last step in the type IV subtype a-b hypersensitivity responseactivation of the intrinsic pathway indicates that external factors, such as UV-A and -B are the trigger. | en_US |
dc.relation | es | |
dc.subject | Odontología | es |
dc.subject | Ciencias de la salud | es |
dc.title | Apoptosis and apoptotic pathway in actinic prurigo by immunohistochemistry | es |
dc.type | journal article | es_ES |
dc.subject.unesco | UNESCO::CIENCIAS MÉDICAS | es |
dc.type.hasVersion | VoR | es_ES |