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dc.contributor.author | Moreno Torres, Marta | |
dc.contributor.author | Jaquenoud, Malika | |
dc.contributor.author | De Virgilio, Claudio | |
dc.date.accessioned | 2023-05-22T07:00:48Z | |
dc.date.available | 2023-05-22T07:00:48Z | |
dc.date.issued | 2015 | |
dc.identifier.citation | Moreno Torres, Marta Jaquenoud, Malika De Virgilio, Claudio 2015 TORC1 controls G1-S cell cycle transition in yeast via Mpk1 and the greatwall kinase pathway Nature Communications 6 8256 1 10 | |
dc.identifier.uri | https://hdl.handle.net/10550/86739 | |
dc.description.abstract | The target of rapamycin complex 1 (TORC1) pathway couples nutrient, energy and hormonal signals with eukaryotic cell growth and division. In yeast, TORC1 coordinates growth with G1-S cell cycle progression, also coined as START, by favouring the expression of G1 cyclins that activate cyclin-dependent protein kinases (CDKs) and by destabilizing the CDK inhibitor Sic1. Following TORC1 downregulation by rapamycin treatment or nutrient limitation, clearance of G1 cyclins and C-terminal phosphorylation of Sic1 by unknown protein kinases are both required for Sic1 to escape ubiquitin-dependent proteolysis prompted by its flagging via the SCFCdc4 (Skp1/Cul1/F-box protein) ubiquitin ligase complex. Here we show that the stabilizing phosphorylation event within the C-terminus of Sic1 requires stimulation of the mitogen-activated protein kinase, Mpk1, and inhibition of the Cdc55 protein phosphatase 2A (PP2ACdc55) by greatwall kinase-activated endosulfines. Thus, Mpk1 and the greatwall kinase pathway serve TORC1 to coordinate the phosphorylation status of Sic1 and consequently START with nutrient availability. | |
dc.language.iso | eng | |
dc.relation.ispartof | Nature Communications, 2015, vol. 6, num. 8256, p. 1-10 | |
dc.subject | Bioquímica | |
dc.subject | Biologia | |
dc.title | TORC1 controls G1-S cell cycle transition in yeast via Mpk1 and the greatwall kinase pathway | |
dc.type | journal article | |
dc.date.updated | 2023-05-22T07:00:48Z | |
dc.identifier.doi | 10.1038/ncomms9256 | |
dc.identifier.idgrec | 158857 | |
dc.rights.accessRights | open access |